Searching journal content for articles similar to Tsai et al. 232 (2): R115.

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  1. Research:
    • Manjunath Ramanjaneya,
    • Bee K Tan,
    • Marcin Rucinski,
    • Mohamed Kawan,
    • Jiamiao Hu,
    • Jaspreet Kaur,
    • Vanlata H Patel,
    • Ludwik K Malendowicz,
    • Hanna Komarowska,
    • Hendrik Lehnert,
    • and Harpal S Randeva
    Nesfatin-1 inhibits proliferation and enhances apoptosis of human adrenocortical H295R cells J Endocrinol 226 (1) 1-11, doi: 10.1530/JOE-14-0496 First published online 13 April 2015
    ...in obesity. Endocrinology 151 3169–3180. (doi:10.1210/en.2009-1358)Wyllie AH, Kerr JF & Currie AR 1973a Cell death in the normal neonatal rat adrenal cortex. Journal of Pathology 111 255–261. (doi:10.1002/path. 1711110406) Wyllie AH, Kerr JF, Macaskill IA & Currie AR 1973b Adrenocortical...
  2. Review:
    • Yolanda Diz-Chaves,
    • Manuel Gil-Lozano,
    • Laura Toba,
    • Juan Fandiño,
    • Hugo Ogando,
    • Lucas C González-Matías,
    • and Federico Mallo
    Stressing diabetes? The hidden links between insulinotropic peptides and the HPA axis J Endocrinol 230 (2) R77-R94, doi: 10.1530/JOE-16-0118 First published online 20 June 2016
    ...contributors to the development of insulin resistance and impaired glucose tolerance (Reaven 1993). Glucocorticoids induce insulin resistance by reducing the expression and phosphorylation of IRS-1 (Sakoda et  al. 2000), thereby impairing the intracellular signalling activated by the IR/IGF-1R. These effects...
  3. Review:
    • Alex Rafacho,
    • Henrik Ortsäter,
    • Angel Nadal,
    • and Ivan Quesada
    Glucocorticoid treatment and endocrine pancreas function: implications for glucose homeostasis, insulin resistance and diabetes J Endocrinol 223 (3) R49-R62, doi: 10.1530/JOE-14-0373 First published online 30 September 2014
    ...knowledge of GCs’ intracellular signalling mechanisms and effects will help to design better GCof GC treatment (van Raalte et al. 2009). Thus, experimental data from human, although of great relevance, fail to totally mimic the conditions of clinical practice (i.e. duration). Elaboration of protocols...
  4. Review:
    • Anne-Marie O'Carroll,
    • Stephen J Lolait,
    • Louise E Harris,
    • and George R Pope
    The apelin receptor APJ: journey from an orphan to a multifaceted regulator of homeostasis J Endocrinol 219 (1) R13-R35, doi: 10.1530/JOE-13-0227 First published online 13 August 2013
    ...distributions of APJ mRNA between mouse and rat. While the significance of this is not known, it may reflect a more extensive role for apelin in mouse pituitary function. Additionally, strong hybridisation can be found in the lung, heart, adrenal cortex, renal medulla, ovary and uterus. These findings confirm...
  5. Thematic Review:
    • Charles A Ducsay and
    • Dean A Myers
    eNOS activation and NO function: Differential control of steroidogenesis by nitric oxide and its adaptation with hypoxia J Endocrinol 210 (3) 259-269, doi: 10.1530/JOE-11-0034 First published online 8 June 2011
    ...to be influenced by NO. This occurs in multiple steroidogenic regions of the adrenal cortex. In adult rat zona glomerulosa, NO clearly inhibited not only basal, but also ACTH and angiotensin II-induced aldosterone production (Hanke et al. 1998). Interestingly, later studies by the same group observed...
  6. PROCEEDINGS OF THE JOINT MEETING OF THE BRITISH ENDOCRINE SOCIETIES J Endocrinol 129 (3 Suppl) NP, doi: 10.1677/joe.0.129S0NP
    ...) plays a key role in determining the final secreted steroid products. The differences in levels of these enzymes are critical when one considers the secretion of zone-specific steroids by the three zones of the adrenal cortex. The relative levels of expression of these enzymes in determining the ability...
  7. PROCEEDINGS OF THE JOINT MEETING OF THE BRITISH ENDOCRINE SOCIETIES J Endocrinol 117 (3 Suppl) NP, doi: 10.1677/joe.0.117S0NP
    ...OF EPITHELIAL CELL PROLIFERATION N.A. Wright, Royal Postgraduate Medical School, Du Cane Road, London and I.C.R.F., Lincoln's Inn Fields, London. Hormones are intimately concerned with the maintenance of tissue mass in target organs such as the adrenal cortex and thyroid. Additionally, hormones...
  8. PROCEEDINGS OF THE JOINT MEETING OF THE BRITISH ENDOCRINE SOCIETIES J Endocrinol 124 (3 Suppl) NP, doi: 10.1677/joe.0.124S0NP
    ...in nerve tissue suggest that ET may also function as a neronal regulator. Prolactin stimulated synthesis of ET-1 has been observed in cultured human breast epithelial cells (hBEP) and peptide promoted intracellular signal transduction in cultured human breast stromal cells (hBST) which were unable...
  9. PROCEEDINGS OF THE JOINT MEETING OF THE BRITISH ENDOCRINE SOCIETIES J Endocrinol 121 (3 Suppl) NP, doi: 10.1677/joe.0.121S0NP
    ...and size of GH pulses; pituitary GH secretion characteristics can be "deconvolved" from such data. GH secretion is modulated by both the gonadal steroid milieu and the nutritional state. Oestrogens and fasting each enhance GH secretion, while it is diminished in obesity. The metabolic role ofGH in adult...
  10. PROCEEDINGS OF THE SOCIETY FOR ENDOCRINOLOGY J Endocrinol 115 (3 Suppl) NP, doi: 10.1677/joe.0.115S0NP
    ...islets. These experiments did not produce clear evidence for a role of chemiosmotic lysis of granules in the insulin secretory mechanism. The granule membrane also contains a Mg-ATPase proton pump and a 29 KDa substrate which is phosphorylated by protein kinase C. A number of other proteins including...
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