Searching journal content for articles similar to Zhao et al. 57 (1): R19.

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  1. Review:
    • T Traboulsi,
    • M El Ezzy,
    • J L Gleason,
    • and S Mader
    Antiestrogens: structure-activity relationships and use in breast cancer treatment J Mol Endocrinol 58 (1) R15-R31, doi: 10.1530/JME-16-0024 first published on 11 October 2016
    ...15–R31 Correspondence should be addressed to S Mader Email [email protected] Key Words f breast cancer f estrogen receptors f antiestrogens f SUMOylation f ubiquitination DOI: 10.1530/JME-16-0024 http://.endocrinology-journals.org © 2017 The authors Printed in Great Britain Published...
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  2. Review:
    • Tram B Doan,
    • J Dinny Graham,
    • and Christine L Clarke
    Emerging functional roles of nuclear receptors in breast cancer J Mol Endocrinol 58 (3) R169-R190, doi: 10.1530/JME-16-0082 first published on 13 January 2017
    ...be mediated through its anti-estrogenic actions as well as its role in regulating the activity of aromatases, the enzymes responsible for the local biosynthesis of estrogens from androgens (Cos et al. 2006). Recent studies using MCF-7 breast cancer xenografts in mice showed that suppression of melatonin...
  3. Research:
    • M Rodríguez-Sanz,
    • N García-Giralt,
    • D Prieto-Alhambra,
    • S Servitja,
    • S Balcells,
    • R Pecorelli,
    • A Díez-Pérez,
    • D Grinberg,
    • I Tusquets,
    • and X Nogués
    CYP11A1 expression in bone is associated with aromatase inhibitor-related bone loss J Mol Endocrinol 55 (1) 69-79, doi: 10.1530/JME-15-0079 first published on 24 June 2015
    ...K, Tournigand C, Decleves X, de Cremoux P et al. 2014 Classification of and risk factors for estrogen deprivation pain syndromes related to aromatase inhibitor treatments in women with breast cancer: a prospective multicenter cohort study. Journal of Pain 15 293–303. (doi:10.1016/j.jpain.2013...
  4. Thematic Review:
    • Evan R Simpson and
    • Kristy A Brown
    Obesity and breast cancer: role of inflammation and aromatase J Mol Endocrinol 51 (3) T51-T59, doi: 10.1530/JME-13-0217 first published on 25 October 2013
    ...primarily to postmenopausal and breast cancer risk is more controversial (Cheraghi et al. ld 2013). This is largely due to overall adiposity rather than ts. More recently, waist-to-hip Thematic Review E R SIMPSON and K A BROWN Obesity, inflammation and aromatase 51 :3 T51–T59and renal failure. What is less...
  5. Review:
    • Caroline H Diep,
    • Andrea R Daniel,
    • Laura J Mauro,
    • Todd P Knutson,
    • and Carol A Lange
    Progesterone action in breast, uterine, and ovarian cancers J Mol Endocrinol 54 (2) R31-R53, doi: 10.1530/JME-14-0252 first published on 13 January 2015
    ...et al. 2010), demonstrate that women taking progestins added to estrogen therapy are at greater risk of developing breast tumors. Unexpectedly, estrogen alone as a single-agent therapy for women who have had a hysterectomy confers protection against breast cancer (hazard ratio 0.77; 95% CI 0...
  6. Thematic Review:
    • Evan Simpson and
    • Richard J Santen
    Celebrating 75 years of oestradiol J Mol Endocrinol 55 (3) T1-T20, doi: 10.1530/JME-15-0128 first published on 5 October 2015
    ...with early-stage breast cancer treated with chemotherapy and an aromatase inhibitor, who exhibited poorer executive function including working memory and concentration (Bender et al. 2015).(Agarwal et al. 1998). This resulted in short stature due to Published by Bioscientifica Ltd. proportion to adiposity...
  7. Review:
    • Russell A Prough,
    • Barbara J Clark,
    • and Carolyn M Klinge
    Novel mechanisms for DHEA action J Mol Endocrinol 56 (3) R139-R155, doi: 10.1530/JME-16-0013 first published on 23 February 2016
    ...)-driven luciferase reporter assay in GT1-7 mouse hypothalamic neuronal cells to a similar extent as 10 nM E2, suggesting ~1000-fold lower affinity (Bruder et al. 1997). DHEA did not appear to be metabolized to E2 in the GT1-7 cells. In MCF-7 and MCF-7SH (an estrogen nonresponsive subline) breast cancer cells...
  8. Research:
    • Yue Wang,
    • Xiu Long Niu,
    • Xiao Qin Guo,
    • Jing Yang,
    • Ling Li,
    • Ye Qu,
    • Cun Xiu Hu,
    • Li Qun Mao,
    • and Dan Wang
    IL6 induces TAM resistance via kinase-specific phosphorylation of ERα in OVCA cells J Mol Endocrinol 54 (3) 351-361, doi: 10.1530/JME-15-0011 first published on 5 May 2015
    ...breast cancer treated with aromatase inhibitors. Oncology 79 55–61. (doi:10.1159/000319540) Murphy LC, Niu Y, Snell L & Watson P 2004 Phospho-serine-118 estrogen receptor-a expression is associated with better disease outcome in women treated with tamoxifen. Clinical Cancer Research 10 5902–5906. (doi:10...
  9. Review:
    • Rosalia C M Simmen,
    • Melissa E Heard,
    • Angela M Simmen,
    • Maria Theresa M Montales,
    • Meera Marji,
    • Samantha Scanlon,
    • and John Mark P Pabona
    The Krüppel-like factors in female reproductive system pathologies J Mol Endocrinol 54 (2) R89-R101, doi: 10.1530/JME-14-0310 first published on 5 February 2015
    ...; ii) the absence of several KLFs in distinct pathologies (e.g. KLF9 and KLF4 in endometrial cancer and endometriosis; KLF9 and KLF11 in endometriosis and leiomyoma) indicates roles for multiple KLFs in maintainLF e Ref Sim Sim Yoo KLF6 (h) Y DiF KLF9 (h) Y/[ Hu Yan Lee Daf Rac Yin Sun Sim Zen Review R...
  10. Articles:
    • SE Bulun,
    • KM Zeitoun,
    • K Takayama,
    • and H Sasano
    Estrogen biosynthesis in endometriosis: molecular basis and clinical relevance J Mol Endocrinol 25 (1) 35-42, doi: 10.1677/jme.0.0250035
    ...that only circulating estradiol, whether secreted by the ovary or formed in the adipose tissue, could exert an estrogenic effect after delivery to target tissues via the bloodstream. Studies on aromatase expression in breast cancer demonstrated that paracrine mechanisms play an important role in estrogen...
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